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Search Results to Jinhu Xiong

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One or more keywords matched the following items that are connected to Xiong, Jinhu

Item TypeName
Academic Article The RANKL distal control region is required for the increase in RANKL expression, but not the bone loss, associated with hyperparathyroidism or lactation in adult mice.
Academic Article Receptor activator of nuclear factor ?B ligand (RANKL) protein expression by B lymphocytes contributes to ovariectomy-induced bone loss.
Academic Article Matrix-embedded cells control osteoclast formation.
Concept RANK Ligand
Academic Article Osteocyte-derived RANKL is a critical mediator of the increased bone resorption caused by dietary calcium deficiency.
Academic Article A DNA segment spanning the mouse Tnfsf11 transcription unit and its upstream regulatory domain rescues the pleiotropic biologic phenotype of the RANKL null mouse.
Academic Article Osteocytes, not Osteoblasts or Lining Cells, are the Main Source of the RANKL Required for Osteoclast Formation in Remodeling Bone.
Academic Article Osteocyte RANKL: new insights into the control of bone remodeling.
Academic Article Cortical bone loss caused by glucocorticoid excess requires RANKL production by osteocytes and is associated with reduced OPG expression in mice.
Academic Article RANKL (Receptor Activator of NF?B Ligand) Produced by Osteocytes Is Required for the Increase in B Cells and Bone Loss Caused by Estrogen Deficiency in Mice.
Academic Article Soluble RANKL contributes to osteoclast formation in adult mice but not ovariectomy-induced bone loss.
Academic Article Osteocyte RANKL is required for cortical bone loss with age and is induced by senescence.
Academic Article Deletion of a putative promoter-proximal Tnfsf11 regulatory region in mice does not alter bone mass or Tnfsf11 expression in vivo.

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  • RANK Ligand